The aim of the present study was to investigate the molecular mechanisms underlying the effects of curcumin on H/R-injured cardiomyocytes. dismutase (SOD) were measured to assess cell injury. Levels of reactive oxygen CEP-37440 varieties (ROS) and apoptosis were evaluated by circulation cytometry. The manifestation levels of Notch intracellular website (NICD) and several downstream genes were … Continue reading The aim of the present study was to investigate the molecular mechanisms underlying the effects of curcumin on H/R-injured cardiomyocytes